Monthly Archives: June 2012

Yeah

When we do a medic discussion i prefer it to be interactive,
To be productive, not just merely sticking to books or to what the doctors say. They may be right or wrong…but i like the discussion to be argumentative, not because we want to point out that our answer is right…Its ok if my answer is wrong, but i defend my answers because that is what i think is right…according to my opinion.everyone is entitled to their own opinions 🙂

I will defend my answer until someone comes along to tell me a reliable answer that i can depend on.

Because i am stubborn like that. Ha ha ha.

A healthy discussion is the way to go!!

Ok there i have said it.
So please be calm .i am not here to fight..i am just being defensive.teeeheeeee

Ecg, ami

There is always confusion around the terms third-degree heart block and AV dissociation. Dr. Chou makes a clear distinction in his book (see Additional Readings section): “The term complete AV block is used when the atrial rate is faster than the ventricular rate, whereas the reverse is true in AV dissociation. In AV block, there is a failure of impulse conduction even though the ventricles are receptive. In AV dissociation, there is an increase in the automaticity of the subsidiary pacemaker, which renders the ventricles functionally refractory to the slower atrial impulses.” These are the definitions we have used in this book.

Look at V1, which is usually the best place to see P waves

Myoglobin levels rise within 1-4 h fr onset of chest pain!!!! The earliest!!

Dont give nitrates in RV INFARCT , SHOCK, AORTIC STENOSIS, HYPOTENSION, CARDIOGENIC SHOCK

In 1st dgree heart block there is ONLY PROLONGATION OF PR BUT IN

2nd degree ( mobitz type 1) there is PROGRESSIVE PROLONGATION OF PR.

MOBITZ TYPE II –> there is constant PR BT OCCASIONALLY, THERE IS WITHOUT A QRS!!!
PLLLLLLLZZZZZZ INGAT SYG OIIIIIII

The triad of right vt infarction( hypotension, jugular venous distension, clear lung fields)

Its just a little crush

AaaaaaaaaAAAAAAAAAAAAAAAAAAAAA!!!!!!!!

My medic crush just replied my message medic questions regarding cardiology.duhh.

Im so happy!!!

Booster dose

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Despite the fact that i am falling in love for cardiology, i dont think its a wise idea to start and reveal my ambition to my parents….just yet.

My parents are almost perfectionist.they are somewhat insatiable to begin with. Even if you do so much,or your best, that just might not be the best for them. Plus, my dad has issues. I dont know why for some reasons he is very likely to underestimate my abilities. But its a motivational drive for me. I take the good side.no doubt, i get hurt at times. But being hurt is good .it keeps you strong.

People may wonder, how are you going to do this for the sake of allah. I am not an excellent candidate to answer that.but basically, if you love your CREATOR, you just want to do the best that you can. So that He will recognize you for doing something.

Just do your very best. Your very2 best!!!Maximum output ok!

Cardiology love!

I am falling head over heels over cardiology.
I am determined to become one!!! Biiznillah…

allah knows best.

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Jejunoileal atresia

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Type I:
intraluminal diaphragm or web may present with windsock anomaly.
NO MESENTERIC DEFECT
NORMAL LENGTH

Type II
Connected by fibrous atretic cord.
NO MESENTERIC DEFECT
NORMAL LENGTH

Type IIIA
NO FIBROUS CORD
VSHAPED MESENTERIC DEFECT
SHORTENED LENGTH

Type IIIB
bowel wraps around a single perfusing vessel( christmas tree app)
LARGE MESENTERIC DEFECT
SIGNIFICANTLY SHORTENED

Type IV
MULTIPLE DEFECTS

Whereas associated anomalies are found in 30-40% of neonates with duodenal atresias, associated anomalies are found in only 10% of neonates with jejunoileal atresias

There is maternal polyhydramnios

RX
RESECTION AND ANASTOMOSIS
TAPERING ENTEROPLASTy(for shortenede bowel)

From emedicine:

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Intestinal atresia type IIIa. A clamp is applied on the distal bowel, and sodium chloride solution is instilled through a purse-string suture to dilate the intestine and diminish the size discrepancy between the two loops to facilitate the anastomosis. The dotted line marks the area of the resection.

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The proximal dilated pouch is transected in a 90° angle to maximize its vascularity, while the distal intestine is transected obliquely to diminish the size discrepancy between the segments.

Malrotation and volvulus neonatorum

Volvulus always associated with malrotation

Two presentations
Acute:bilious vomiting(metabolic acidosis) The volvulus may compromise blood flow leading to ischemia hence tender rigid abdomen

We fear from massive loss of small intestine if neglected…he may not survive…or the lesser catastrophe is the SHORT GUT SYNDROME

Chr: chr intmt abd pain, constipation.

DOUBLE BUBBLE SIGN

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Normally SMV IS ON THE RIGHT. IF THE REVERSE HAPPENS, SUSPECT MALROTATION.(BY U/S!!!!!)
DILATED STOMACh AND DUODENUM WITH TWISTING OF THE MIDGUT LOOP: applepeel, twisted ribbon, corkscrew

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Do ladds procedure:
Untwist
Divide( ladds band)
appendectomy

Neonatal duodenal obstruction

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SEARCH FOR ASSOCIATED ANOMALIES!

Normal or scaphoid abdomen is obs is proximal!

FIRST OF ALL, RESUSCITATE AND DECOMPRESSION

ITS DANGEROUS!!! WHY??
close proximity to the ampulla of vater & pancreatic blood supply.

Rx: DUODENODUODENOSTOMY AND DUODENOJEJUNOSTOMY. IF THERE IS MALROT- LADDS PROCEDURE
we can also do DIAMON SHAPED ANASTOMOSIS BECAUSE THE DIAMETER OF THE PROXIMAL AND DISTAL PART TO THE OBSTRUCTION IS NOT PROPORTIANATE.

In duodenal web, do DUODENOTOMY. BE CAREFUL OF OTHER DEFECTS AND AMPULLA OF VATER.

?

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About ladds band(excerpt from emedicine)

abnormal cecal peritoneal bands that cross over the duodenum in the right upper quadrant may be found in cases of malrotation. These abnormal cecal attachments are called Ladd bands. In both nonrotation and incomplete rotation, cecal bands may cause obstruction and ischemia before birth, leading to congenital duodenal atresia or stenosis

In many cases, the abnormal Ladd bands cause only partial and intermittent obstruction, which can manifest as intermittent episodes of bilious vomiting and/or feeding intolerance.

I5

Previously healthy baby

Red currant jelly stool is a late sign..dont sit and wait for that to happen

Not so hi mortality rate but we fear of transmural gangrene

Commonest age…age of weaning 4m-10m

If more than 2 y, suspect a secondary cause..the most common 2ry cause is ileocecal lymphoma and meckels

Important signs:
Dance s
Target s
Cobra head s
Air fluid level
Pseudokidney s

25% of abdominal surgical emergencies less than 5, EXCEEDING APPENDICITIS

ANY CHILD W BILIOUS VOMITING, CONSIDER IT A SURGICAL DISEASEUNTIL PROVED OTHERWISE

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IHPS

More common in males

First male

Hypochloraemia, hyponatremia, hypokalemia metabolic alkalosis

U/s! Thickness >4mm, length >16 mm

Dehydration, failure to thrive, HUNGER AFTER FEEDING,OLIVE MASS, PROJECTILE, NON BILIOUS VOMITING, persistent and progresses with time.

IT IS NOT CONGENITAL HENCE IT NEVER PRESENTS before 2weeks of life. It takes time for the circular muscle to undergo HYPERTROPHY AND HYPERPLASIA ( >2wks- 8wks)

DDX OF NON BILIOUS VOMITING: physiological GERD/ proximal to ampulla of vater duodenal obstruction/ errors in feeding/ NON SPECIFIC MARKER OF ILLNESS

TREATMENT: RESUSCITATE FIRST, THIS IS NOT AN EMERGENCY.
DEFINITIVE TREATMENT= RAMSTEDT PYLOROMYOTOMY

Important! Look for target sign.
String sign.

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